Licensed U.S. pharmacy
If offered, each prescription would be filled through a licensed compounding pharmacy.

Cell studies measured lower NF-κB and MAPK activation. Mouse colitis studies measured less inflammation and earlier recovery.
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K · P · V
KPV is lysine-proline-valine. In cell experiments it reduced TNF-α-driven NF-κB and MAPK signaling. In mouse colitis models, researchers measured earlier recovery and less colonic inflammation.

Coming soon
If KPV launches, the planned 20-day format will be dispensed by a licensed U.S. compounding pharmacy. The patient-specific label will identify the formulation, strength, storage, and beyond-use date.

Why the gut research exists
PepT1Researchers identified PepT1-mediated uptake in intestinal and immune cells. Inside the cell, KPV reduced two major inflammatory signal networks: NF-κB and MAPK.
What the mouse studies found
Across DSS colitis and transfer colitis, KPV-treated mice regained weight earlier and showed less inflammation in colon tissue. One oral KPV experiment used five mice per group and measured weight, myeloperoxidase activity, and histology.

What has been studied
Published KPV evidence comes from cell experiments and animal models. No human KPV exposure, safety, or efficacy studies were identified by any route. The planned Rebody format is an injection; the published studies do not establish results for that format.
Read the evidence review ↗The Rebody difference
If offered, each prescription would be filled through a licensed compounding pharmacy.
The formulation, strength, storage instructions, and beyond-use date would appear on the vial.
Ordering would begin with Rebody intake and a prescription decision—not a research-marketplace checkout.
Orders are closed
We'll send the final price, formulation, directions, states served, sourcing details, and ordering date when KPV becomes available.
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